https://nova.newcastle.edu.au/vital/access/manager/Index en-au 5 https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:48569 Wed 22 Mar 2023 15:26:43 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:20390 Wed 11 Apr 2018 16:32:40 AEST ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:18827 Wed 11 Apr 2018 13:01:16 AEST ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:27705 0.9 between SNPs). Although SNPs associated with deep intracerebral hemorrhage did not reach our significance threshold for association with lacunar ischemic stroke (lead SNP OR 1.10, 95% CI 1.03-1.18, p = 0.0073), and with white matter hyperintensity volume in symptomatic ischemic stroke patients (lead SNP OR 1.07, 95% CI 1.01-1.13, p = 0.016), the direction of association was the same. There was no convincing evidence of association with white matter hyperintensities in population-based studies or with non-small vessel disease cerebrovascular phenotypes. Conclusions: Our results indicate an association between common variation in the COL4A2 gene and symptomatic small vessel disease, particularly deep intracerebral hemorrhage. These findings merit replication studies, including in ethnic groups of non-European ancestry.]]> Tue 21 Jul 2020 09:43:56 AEST ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:11142 Sat 24 Mar 2018 08:10:28 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:27208 Sat 24 Mar 2018 07:32:26 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:46168 Fri 11 Nov 2022 19:54:47 AEDT ]]>