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Intracranial pressure elevation post-stroke: Mechanisms and consequences

Leakage beyond the primary lesion: A temporal analysis of cerebrovascular dysregulation at sites of hippocampal secondary neurodegeneration following cortical photothrombotic stroke

Tuberous Sclerosis Complex‐1 (TSC1) contributes to selective neuronal vulnerability in Alzheimer’s Disease

Growth Hormone Increases BDNF and mTOR Expression in Specific Brain Regions after Photothrombotic Stroke in Mice

Neurovascular coupling mechanisms in health and neurovascular uncoupling in Alzheimer's disease

Decreased Intracranial Pressure Elevation and Cerebrospinal Fluid Outflow Resistance: A Potential Mechanism of Hypothermia Cerebroprotection Following Experimental Stroke

Short-duration hypothermia completed prior to reperfusion prevents intracranial pressure elevation following ischaemic stroke in rats

Ultra-Short Duration Hypothermia Prevents Intracranial Pressure Elevation Following Ischaemic Stroke in Rats

Rapamycin induces an eNOS (Endothelial Nitric Oxide Synthase) dependent increase in brain collateral perfusion in Wistar and spontaneously hypertensive Rats

The rise of pericytes in neurovascular research

Intracranial pressure and collateral blood flow

Intracranial pressure elevation 24 h after ischemic stroke in aged rats is prevented by early, short hypothermia treatment

Ischemic penumbra as a trigger for intracranial pressure rise: a potential cause for collateral failure and infarct progression?

Intracranial pressure elevation after ischemic stroke in rats: cerebral edema is not the only cause, and short-duration mild hypothermia is a highly effective preventive therapy

Intracranial pressure elevation reduces flow through collateral vessels and the penetrating arterioles they supply. A possible explanation for 'collateral failure' and infarct expansion after ischemic stroke

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