Corticosterone administration alters white matter tract structure and reduces gliosis in the sub-acute phase of experimental stroke
Exploring how low oxygen post conditioning improves stroke-induced cognitive impairment: a consideration of amyloid-beta loading and other mechanisms
- Zhao, Zidan, Hood, Rebecca, Ong, Lin Kooi, Pietrogrande, Giovanni, Sanchez Bezanilla, Sonia, Warren, Kirby E., Ilicic, Marina, Kluge, Murielle G., TeBay, Clifford, Ottersen, Ole P., Johnson, Sarah J., Nilsson, Michael, Walker, Frederick R.
Growth hormone improves cognitive function after experimental stroke
- Ong, Lin Kooi, Chow, Wei Zhen, Walker, Frederick R., Nilsson, Michael, Isgaard, Jörgen, Tebay, Clifford, Kluge, Murielle, Pietrogrande, Giovanni, Zalewska, Katarzyna, Crock, Patricia, Åberg, Daivd, Bivard, Andrew, Johnson, Sarah J.
Involvement of microglia activation in the development of CNS diseases
Low oxygen post conditioning as an efficient non-pharmacological strategy to promote motor function after stroke
Low oxygen post conditioning prevents thalamic secondary neuronal loss caused by excitotoxicity after cortical stroke
Sustained administration of corticosterone at stress-like levels after stroke suppressed glial reactivity at sites of thalamic secondary neurodegeneration
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