https://nova.newcastle.edu.au/vital/access/manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:37075 vs 21.4 (5.1) cm³; P = 0.027) despite no significant difference in body weight. Despite having a smaller TRV, there was no significant difference in eGFR between Indigenous and Non-indigenous neonates (47.8 [43.2-50.4] vs 46.2 [42.6-53.3] ml/min per 1.73 m²; P = 0.986). These infants achieve similar eGFR through hyperfiltration, which likely increases their future risk of CKD. There was no difference in microalbumin-creatinine ratio. Female Indigenous neonates, however, had significantly smaller TRV compared with Indigenous male neonates (15.9 (3.6) vs 20.6 (3.6) cm³; P = 0.006), despite no difference in eGFR, birth weight, gestational age, and weight at term corrected. Conclusion: The difference in TRV is likely to be an important risk factor for the difference in morbidity and mortality from renal disease reported between male and female Indigenous adults.]]> Wed 15 Dec 2021 16:07:38 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:11019 Wed 11 Apr 2018 14:49:03 AEST ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:13652 Wed 11 Apr 2018 13:13:17 AEST ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:32578 Thu 21 Jun 2018 11:53:16 AEST ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:34951 Thu 09 Dec 2021 11:04:52 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:13370 Sat 24 Mar 2018 10:37:29 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:5186 Sat 24 Mar 2018 07:47:48 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:5474 Sat 24 Mar 2018 07:47:06 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:5510 Sat 24 Mar 2018 07:46:39 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:28160 Sat 24 Mar 2018 07:36:34 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:30838 Sat 24 Mar 2018 07:33:55 AEDT ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:34950 Mon 27 May 2019 12:39:55 AEST ]]> https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:30481 37 completed wk) admitted to the neonatal unit with minor neonatal conditions were recruited into the control group. Complete data sets were available in 91 premature neonates and during the same period, 56 term neonates were recruited as the control. The median birth weight (preterm babies) was 930 g (780-1220 g), and the mean gestational age was 27.0 wk (2.1 wk). Total renal volume (TRV) increased from 14.6 (4.3) cm 3 to 20.5 (5.3) cm 3 from 32 to 37 wk PMA. During the same period, the total renal parenchyma (TRP) thickness increased from 1.6 (0.3) cm to 1.8 (0.3) cm. At 37 wk PMA, ex-premature neonates have a significantly smaller total renal volume (20.5 [5.3] versus 25.9 [6.4] cm 3 ; p < 0.001) and total renal parenchyma thickness (1.8 [0.3] versus 2.0 [0.2] cm; p = 0.015) compared with term (control) neonates. However, premature neonates at 37 wk PMA have a larger TRP:TRV ratio compared with term neonates (0.09 [0.02] versus 0.0 8 [0.02] cm -2 ; p < 0.001). Reduced nephron endowment as a result of prematurity may cause the remaining nephrons to undergo compensatory glomerulomegaly and we postulate this is the reason for the observed differences. Ultrasound imaging of the renal parenchyma shows promise in assessing the effects of prematurity on the developing kidney.]]> Fri 01 Apr 2022 09:21:26 AEDT ]]>