https://nova.newcastle.edu.au/vital/access/manager/Index en-au 5 A cell cycle-associated change in Ca²⁺ releasing activity leads to the generation of Ca²⁺ transients in mouse embryos during the first mitotic division https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6547 Wed 11 Apr 2018 15:10:36 AEST ]]> A novel mechanism controls the Ca²⁺ oscillations triggered by activation of ascidian eggs and has an absolute requirement for Cdk1 activity https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6552 Wed 11 Apr 2018 13:43:05 AEST ]]> How eggs arrest at metaphase II: MPF stabilisation plus APC/C inhibition equals Cytostatic Factor https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6549 Wed 11 Apr 2018 10:43:58 AEST ]]> The passage of Ca²⁺ and fluorescent markers between the sperm and egg after fusion in the mouse https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6548 Wed 11 Apr 2018 10:37:33 AEST ]]> A cytosolic sperm protein factor mobilizes Ca²⁺ from intracellular stores by activating multiple Ca²⁺ release mechanisms independently of low molecular weight messengers https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6596 Wed 11 Apr 2018 09:57:51 AEST ]]> Control of sarcoplasmic reticulum Ca²⁺ release by stochastic RyR gating within a 3D model of the cardiac dyad and importance of induction decay for CICR termination https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:20204 Sat 24 Mar 2018 08:06:48 AEDT ]]> Spatial quantification of cytosolic Ca²⁺ accumulation in nonexcitable cells: an analytical study https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:19117 Sat 24 Mar 2018 07:55:52 AEDT ]]> Different Ca²⁺-releasing abilities of sperm extracts compared with tissue extracts and phospholipase C isoforms in sea urchin egg homogenate and mouse eggs https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6571 Sat 24 Mar 2018 07:49:16 AEDT ]]> Meiotic and mitotic Ca²⁺ oscillations affect cell composition in resulting blastocysts https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6585 Sat 24 Mar 2018 07:49:10 AEDT ]]> A comparison of sperm- and IP₃-induced Ca²⁺ release in activated and aging mouse oocytes https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6584 Sat 24 Mar 2018 07:49:10 AEDT ]]> Ca²⁺ release and the development of Ca²⁺ release mechanisms during oocyte maturation: a prelude to fertilization https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6540 Sat 24 Mar 2018 07:48:19 AEDT ]]> Ionomycin, thapsigargin, ryanodine, and sperm induced Ca²⁺ release increase during meiotic maturation of mouse oocytes https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6541 Sat 24 Mar 2018 07:48:19 AEDT ]]> Ca²⁺ oscillations in the activation of the egg and development of the embryo in mammals https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6594 Sat 24 Mar 2018 07:46:22 AEDT ]]> On the search for the sperm oscillogen https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:6600 Sat 24 Mar 2018 07:45:36 AEDT ]]> Beta-adrenergic stimulation increases RyR2 activity via intracellular Ca²⁺ and Mg²⁺ regulation https://nova.newcastle.edu.au/vital/access/manager/Repository/uon:13304 a and maximum levels of cytoplasmic Ca²⁺ activation site were not affected by ß-adrenergic stimulation. Our RyR2 gating model was fitted to the single channel data. It predicted that in diastole, ß-adrenergic stimulation is mediated by 1) increasing the activating potency of Ca²⁺ binding to the luminal Ca²⁺ site and decreasing its affinity for luminal Mg²⁺ and 2) decreasing affinity of the low-affinity Ca²⁺/Mg²⁺ cytoplasmic inhibition site. However in systole, ß-adrenergic stimulation is mediated mainly by the latter.]]> Mon 15 Jul 2024 14:47:49 AEST ]]>