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Zhang, Xu Dong

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Nucleotide excision repair gene expression after cisplatin treatment in melanoma 92 89 0
Metabolic approaches to treatment of melanoma 89 81 3
Small molecular weight variants of p53 are expressed in human melanoma cells and are induced by the DNA-damaging agent cisplatin 96 91 0
Palmitoylation of CD36/FAT regulates the rate of its post-transcriptional processing in the endoplasmic reticulum 72 63 0
Human melanoma cells under endoplasmic reticulum stress acquire resistance to microtubule-targeting drugs through XBP-1-mediated activation of Akt 65 64 0
Up-regulation of Mcl-1 is critical for survival of human melanoma cells upon endoplasmic reticulum stress 89 82 0
Inhibition of MEK blocks GRP78 up-regulation and enhances apoptosis induced by ER stress in gastric cancer cells 81 74 0
Expression of glucose-regulated stress protein GRP78 is related to progression of melanoma 69 59 0
Activation of Jun N-terminal kinase is a mediator of vincristine-induced apoptosis of melanoma cells 181 174 0
Involvement of endoplasmic reticulum stress in docetaxel-induced JNK-dependent apoptosis of human melanoma 79 75 0
Inhibition of apoptosis facilitates necrosis induced by cisplatin in gastric cancer cells 84 76 0
Contrasting effects of Nutlin-3 on TRAIL- and Docetaxel-induced Apoptosis due to upregulation of TRAIL-R2 and Mcl-1 in human melanoma cells 123 123 0
2-Deoxy-D-glucose enhances TRAIL-induced apoptosis in human melanoma cells through XBP-1-mediated up-regulation of TRAIL-R2 123 112 5
TRAIL-induced apoptosis of human melanoma cells involves activation of caspase-4 40 36 0
Adaptation to ER stress as a driver of malignancy and resistance to therapy in human melanoma 62 54 2
Human melanoma cells under endoplasmic reticulum stress are more susceptible to apoptosis induced by the BH3 mimetic obatoclax 86 84 0
Glucose-regulated protein 78 antagonizes cisplatin and adriamycin in human melanoma cells 96 87 0
Inhibition of endoplasmic reticulum stress-induced apoptosis of melanoma cells by the ARC protein 52 51 0
Treatment combinations targeting apoptosis to improve immunotherapy of melanoma 53 51 1
Human melanoma cells selected for resistance to apoptosis by prolonged exposure to tumor necrosis factor-related apoptosis-inducing ligand are more vulnerable to necrotic cell death induced by cisplatin 34 32 0
Progression in melanoma is associated with decreased expression of death receptors for tumor necrosis factor–related apoptosis-inducing ligand 59 52 0
Lactate dehydrogenase 5 expression in melanoma increases with disease progression and is associated with expression of Bcl-XL and Mcl-1, but not Bcl-2 proteins 60 59 0
Translational control of tumor necrosis factor-related apoptosis-inducing ligand death receptor expression in melanoma cells 41 41 0
Overcoming resistance to apoptosis in cancer therapy 41 41 0
Melanoma cell sensitivity to docetaxal-induced apoptosis is determined by class III β-tubulin levels 55 50 0
MicroRNA-149*, a p53-responsive microRNA, functions as an oncogenic regulator in human melanoma 13 13 0
Applications of nanotechnology for melanoma treatment, diagnosis, and theranostics 6 6 0
PI(4,5)P2 5-phosphatase A regulates PI3K/Akt signalling and has a tumour suppressive role in human melanoma 5 4 1
Regulators of global genome repair do not respond to DNA damaging therapy but correlate with survival in melanoma 4 4 0
OBATOCLAX and ABT-737 induce ER stress responses in human melanoma cells that limit induction of apoptosis 4 4 0
Suppression of endoplasmic reticulum stress-induced invasion and migration of breast cancer cells through the downregulation of heparanase 2 2 0
Loss of PI(4,5)P₂ 5-phosphatase A contributes to resistance of human melanoma cells to RAF/MEK inhibitors 2 2 0
Dual processing of FAT1 cadherin protein by human melanoma cells generates distinct protein products 187 168 14
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Zhang, Xu Dong

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