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Zhang, Xu Dong

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Nucleotide excision repair gene expression after cisplatin treatment in melanoma 103 99 0
Metabolic approaches to treatment of melanoma 93 85 4
Small molecular weight variants of p53 are expressed in human melanoma cells and are induced by the DNA-damaging agent cisplatin 104 99 0
Palmitoylation of CD36/FAT regulates the rate of its post-transcriptional processing in the endoplasmic reticulum 76 67 0
Human melanoma cells under endoplasmic reticulum stress acquire resistance to microtubule-targeting drugs through XBP-1-mediated activation of Akt 90 89 0
Inhibition of MEK blocks GRP78 up-regulation and enhances apoptosis induced by ER stress in gastric cancer cells 93 86 0
Expression of glucose-regulated stress protein GRP78 is related to progression of melanoma 74 64 0
Activation of Jun N-terminal kinase is a mediator of vincristine-induced apoptosis of melanoma cells 189 182 0
Involvement of endoplasmic reticulum stress in docetaxel-induced JNK-dependent apoptosis of human melanoma 97 93 0
Inhibition of apoptosis facilitates necrosis induced by cisplatin in gastric cancer cells 106 98 0
Contrasting effects of Nutlin-3 on TRAIL- and Docetaxel-induced Apoptosis due to upregulation of TRAIL-R2 and Mcl-1 in human melanoma cells 127 127 0
2-Deoxy-D-glucose enhances TRAIL-induced apoptosis in human melanoma cells through XBP-1-mediated up-regulation of TRAIL-R2 143 132 6
TRAIL-induced apoptosis of human melanoma cells involves activation of caspase-4 40 36 0
Adaptation to ER stress as a driver of malignancy and resistance to therapy in human melanoma 67 59 3
Human melanoma cells under endoplasmic reticulum stress are more susceptible to apoptosis induced by the BH3 mimetic obatoclax 111 109 0
Glucose-regulated protein 78 antagonizes cisplatin and adriamycin in human melanoma cells 100 91 0
Inhibition of endoplasmic reticulum stress-induced apoptosis of melanoma cells by the ARC protein 57 56 0
Treatment combinations targeting apoptosis to improve immunotherapy of melanoma 55 53 2
Human melanoma cells selected for resistance to apoptosis by prolonged exposure to tumor necrosis factor-related apoptosis-inducing ligand are more vulnerable to necrotic cell death induced by cisplatin 37 34 0
Progression in melanoma is associated with decreased expression of death receptors for tumor necrosis factor–related apoptosis-inducing ligand 74 65 0
Lactate dehydrogenase 5 expression in melanoma increases with disease progression and is associated with expression of Bcl-XL and Mcl-1, but not Bcl-2 proteins 66 65 0
Translational control of tumor necrosis factor-related apoptosis-inducing ligand death receptor expression in melanoma cells 44 44 0
Overcoming resistance to apoptosis in cancer therapy 49 49 0
Melanoma cell sensitivity to docetaxal-induced apoptosis is determined by class III β-tubulin levels 64 59 0
MicroRNA-149*, a p53-responsive microRNA, functions as an oncogenic regulator in human melanoma 38 38 1
Applications of nanotechnology for melanoma treatment, diagnosis, and theranostics 11 11 1
PI(4,5)P2 5-phosphatase A regulates PI3K/Akt signalling and has a tumour suppressive role in human melanoma 146 143 1
Regulators of global genome repair do not respond to DNA damaging therapy but correlate with survival in melanoma 48 47 1
OBATOCLAX and ABT-737 induce ER stress responses in human melanoma cells that limit induction of apoptosis 52 51 0
Low expression of microRNA-146b-5p and microRNA-320d predicts poor outcome of large B-cell lymphoma treated with cyclophosphamide, doxorubicin, vincristine, and prednisone 7 5 0
Insulin induces drug resistance in melanoma through activation of the PI3K/Akt pathway 17 15 0
Antiproliferative effects of continued mitogen-activated protein kinase pathway inhibition following acquired resistance to BRAF and/or MEK inhibition in melanoma 30 30 0
The emerging important role of microRNAs in the pathogenesis, diagnosis and treatment of human cancers 2 2 0
MEK-independent survival of B-RAFV600E melanoma cells selected for resistance to apoptosis induced by the RAF inhibitor PLX4720 11 9 0
Evidence for upregulation of Bim and the splicing factor SRp55 in melanoma cells from patients treated with selective BRAF inhibitors 5 4 0
Suppression of endoplasmic reticulum stress-induced invasion and migration of breast cancer cells through the downregulation of heparanase 4 4 0
Loss of PI(4,5)P₂ 5-phosphatase A contributes to resistance of human melanoma cells to RAF/MEK inhibitors 24 24 0
Dual processing of FAT1 cadherin protein by human melanoma cells generates distinct protein products 240 219 15
Up-regulation of Mcl-1 is critical for survival of human melanoma cells upon endoplasmic reticulum stress 100 89 0
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